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co treatment  (TargetMol)


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    TargetMol co treatment
    Co Treatment, supplied by TargetMol, used in various techniques. Bioz Stars score: 99/100, based on 69 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/co treatment/product/TargetMol
    Average 99 stars, based on 69 article reviews
    co treatment - by Bioz Stars, 2026-03
    99/100 stars

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    RNA-seq and the effects of Mdivi-1, Fer-1, and <t>ML385</t> on neuronal apoptosis and reactive oxygen species (ROS) levels in HT22 neurons. ( a ) Microscopic images of HT22 cells (× 20 magnification). ( b ) Volcano plot of the differentially expressed genes (DEGs) between the control and oxygen–glucose deprivation (OGD) model cells. Blue represents significantly downregulated genes, and red represents significantly upregulated genes. ( c ) GO terms associated with the DEGs. ( d ) KEGG pathway enrichment analysis, highlighting the ferroptosis signaling pathway (red arrow). ( e ) Fluorescence double staining for caspase-3 (green) and NeuN (red). ( f ) Statistical analysis of the relative intensity of caspase-3 fluorescence. ( g , h ) ROS levels in the different groups. ( i , j ) Representative western blot bands for cleaved caspase-3 in HT22 cells. * P < 0.05 vs . the control group, # P < 0.05 vs . the OGD group, ^ P < 0.05 vs . the Mdivi-1 group, & P < 0.05 vs . the Fer-1 group
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    RNA-seq and the effects of Mdivi-1, Fer-1, and <t>ML385</t> on neuronal apoptosis and reactive oxygen species (ROS) levels in HT22 neurons. ( a ) Microscopic images of HT22 cells (× 20 magnification). ( b ) Volcano plot of the differentially expressed genes (DEGs) between the control and oxygen–glucose deprivation (OGD) model cells. Blue represents significantly downregulated genes, and red represents significantly upregulated genes. ( c ) GO terms associated with the DEGs. ( d ) KEGG pathway enrichment analysis, highlighting the ferroptosis signaling pathway (red arrow). ( e ) Fluorescence double staining for caspase-3 (green) and NeuN (red). ( f ) Statistical analysis of the relative intensity of caspase-3 fluorescence. ( g , h ) ROS levels in the different groups. ( i , j ) Representative western blot bands for cleaved caspase-3 in HT22 cells. * P < 0.05 vs . the control group, # P < 0.05 vs . the OGD group, ^ P < 0.05 vs . the Mdivi-1 group, & P < 0.05 vs . the Fer-1 group
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    RNA-seq and the effects of Mdivi-1, Fer-1, and <t>ML385</t> on neuronal apoptosis and reactive oxygen species (ROS) levels in HT22 neurons. ( a ) Microscopic images of HT22 cells (× 20 magnification). ( b ) Volcano plot of the differentially expressed genes (DEGs) between the control and oxygen–glucose deprivation (OGD) model cells. Blue represents significantly downregulated genes, and red represents significantly upregulated genes. ( c ) GO terms associated with the DEGs. ( d ) KEGG pathway enrichment analysis, highlighting the ferroptosis signaling pathway (red arrow). ( e ) Fluorescence double staining for caspase-3 (green) and NeuN (red). ( f ) Statistical analysis of the relative intensity of caspase-3 fluorescence. ( g , h ) ROS levels in the different groups. ( i , j ) Representative western blot bands for cleaved caspase-3 in HT22 cells. * P < 0.05 vs . the control group, # P < 0.05 vs . the OGD group, ^ P < 0.05 vs . the Mdivi-1 group, & P < 0.05 vs . the Fer-1 group
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    Image Search Results


    RNA-seq and the effects of Mdivi-1, Fer-1, and ML385 on neuronal apoptosis and reactive oxygen species (ROS) levels in HT22 neurons. ( a ) Microscopic images of HT22 cells (× 20 magnification). ( b ) Volcano plot of the differentially expressed genes (DEGs) between the control and oxygen–glucose deprivation (OGD) model cells. Blue represents significantly downregulated genes, and red represents significantly upregulated genes. ( c ) GO terms associated with the DEGs. ( d ) KEGG pathway enrichment analysis, highlighting the ferroptosis signaling pathway (red arrow). ( e ) Fluorescence double staining for caspase-3 (green) and NeuN (red). ( f ) Statistical analysis of the relative intensity of caspase-3 fluorescence. ( g , h ) ROS levels in the different groups. ( i , j ) Representative western blot bands for cleaved caspase-3 in HT22 cells. * P < 0.05 vs . the control group, # P < 0.05 vs . the OGD group, ^ P < 0.05 vs . the Mdivi-1 group, & P < 0.05 vs . the Fer-1 group

    Journal: Molecular Neurobiology

    Article Title: Inhibition of DRP1-mediated Mitochondrial Fission and NRF2/HO-1/GPX4-mediated Ferroptosis by Mdivi-1 Protects Against Vascular Cognitive Impairment

    doi: 10.1007/s12035-025-05484-2

    Figure Lengend Snippet: RNA-seq and the effects of Mdivi-1, Fer-1, and ML385 on neuronal apoptosis and reactive oxygen species (ROS) levels in HT22 neurons. ( a ) Microscopic images of HT22 cells (× 20 magnification). ( b ) Volcano plot of the differentially expressed genes (DEGs) between the control and oxygen–glucose deprivation (OGD) model cells. Blue represents significantly downregulated genes, and red represents significantly upregulated genes. ( c ) GO terms associated with the DEGs. ( d ) KEGG pathway enrichment analysis, highlighting the ferroptosis signaling pathway (red arrow). ( e ) Fluorescence double staining for caspase-3 (green) and NeuN (red). ( f ) Statistical analysis of the relative intensity of caspase-3 fluorescence. ( g , h ) ROS levels in the different groups. ( i , j ) Representative western blot bands for cleaved caspase-3 in HT22 cells. * P < 0.05 vs . the control group, # P < 0.05 vs . the OGD group, ^ P < 0.05 vs . the Mdivi-1 group, & P < 0.05 vs . the Fer-1 group

    Article Snippet: The remaining 32 rats were randomly assigned to the following four groups ( n = 8/group): Sham, VCI, Mdivi-1 (VCI + Mdivi-1 [No. HY-15886, Med Chem Express, Shanghai, China]) [ ], and Mdivi-1 + ML385 co-treatment (VCI + Mdivi-1 + ML385 [No. HY-100523, Med Chem Express, Shanghai, China]) [ ].

    Techniques: RNA Sequencing, Control, Fluorescence, Double Staining, Western Blot

    The effects of Mdivi-1 on mitochondrial ultrastructure and ATP levels. ( a ) Representative TOMM20 (green) and DHE (red) immunofluorescence staining in the hippocampus. ( b, c ) Quantitative analysis of relative fluorescence intensity. ( d ) Hippocampal neurons from sham-operated control rats had normal organelles. Apoptotic neurons with nuclear pyknosis could be seen in the VCI group. In the Mdivi-1 + ML385 co-treatment group, the cytoplasm was edematous, and mitochondria within it were swollen and exhibited damaged cristae. Representative neuronal ultrastructure deterioration: severe mitochondrial swelling, cristae disruption, and vacuolation, accompanied by swollen endoplasmic reticulum and reduced organelle number. Scale bar, 1 μm. ( e ) Statistical analysis of relative mitochondrial damage. ( f ) Statistical analysis of the relative ATP levels ( n = 4, * P < 0.05, ** P < 0.001. Nu, nucleus; Mit, mitochondrion; ER, endoplasmic reticulum)

    Journal: Molecular Neurobiology

    Article Title: Inhibition of DRP1-mediated Mitochondrial Fission and NRF2/HO-1/GPX4-mediated Ferroptosis by Mdivi-1 Protects Against Vascular Cognitive Impairment

    doi: 10.1007/s12035-025-05484-2

    Figure Lengend Snippet: The effects of Mdivi-1 on mitochondrial ultrastructure and ATP levels. ( a ) Representative TOMM20 (green) and DHE (red) immunofluorescence staining in the hippocampus. ( b, c ) Quantitative analysis of relative fluorescence intensity. ( d ) Hippocampal neurons from sham-operated control rats had normal organelles. Apoptotic neurons with nuclear pyknosis could be seen in the VCI group. In the Mdivi-1 + ML385 co-treatment group, the cytoplasm was edematous, and mitochondria within it were swollen and exhibited damaged cristae. Representative neuronal ultrastructure deterioration: severe mitochondrial swelling, cristae disruption, and vacuolation, accompanied by swollen endoplasmic reticulum and reduced organelle number. Scale bar, 1 μm. ( e ) Statistical analysis of relative mitochondrial damage. ( f ) Statistical analysis of the relative ATP levels ( n = 4, * P < 0.05, ** P < 0.001. Nu, nucleus; Mit, mitochondrion; ER, endoplasmic reticulum)

    Article Snippet: The remaining 32 rats were randomly assigned to the following four groups ( n = 8/group): Sham, VCI, Mdivi-1 (VCI + Mdivi-1 [No. HY-15886, Med Chem Express, Shanghai, China]) [ ], and Mdivi-1 + ML385 co-treatment (VCI + Mdivi-1 + ML385 [No. HY-100523, Med Chem Express, Shanghai, China]) [ ].

    Techniques: Immunofluorescence, Staining, Fluorescence, Control, Disruption